Search results (13)
« Back to PublicationsFoxO1-zDHHC4-CD36 S-Acylation Axis Drives Metabolic Dysfunction in Diabetes.
Journal article
Dennis KMJH. et al, (2025), Circ Res, 136, 1545 - 1560
Tissue-specific differences in the assembly of mitochondrial Complex I are revealed by a novel ENU mutation in ECSIT.
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Nicol T. et al, (2023), Cardiovasc Res, 119, 2213 - 2229
Cardiomyocyte tetrahydrobiopterin synthesis regulates fatty acid metabolism and susceptibility to ischaemia-reperfusion injury.
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Chu SM. et al, (2023), Exp Physiol, 108, 874 - 890
Chronically elevated branched chain amino acid levels are pro-arrhythmic.
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Portero V. et al, (2022), Cardiovasc Res, 118, 1742 - 1757
Isolation and In vitro Culture of Bone Marrow-Derived Macrophages for the Study of NO-Redox Biology.
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Diotallevi M. et al, (2022), J Vis Exp
Tissue Proteome of 2-Hydroxyacyl-CoA Lyase Deficient Mice Reveals Peroxisome Proliferation and Activation of ω-Oxidation.
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Khalil Y. et al, (2022), Int J Mol Sci, 23
Itaconate as an inflammatory mediator and therapeutic target in cardiovascular medicine.
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CRABTREE M. et al, (2021), Biochemical Society Transactions
Linking the FTO obesity rs1421085 variant circuitry to cellular, metabolic, and organismal phenotypes in vivo.
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Laber S. et al, (2021), Sci Adv, 7
Isolation and culture of murine bone marrow-derived macrophages for nitric oxide and redox biology.
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Bailey JD. et al, (2020), Nitric Oxide, 100-101, 17 - 29
Modification of an aggressive model of Alport Syndrome reveals early differences in disease pathogenesis due to genetic background.
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Falcone S. et al, (2019), Sci Rep, 9
Nitric oxide modulates metabolic remodelling in inflammatory macrophages through TCA cycle regulation and itaconate accumulation
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CRABTREE M. et al, (2019), Cell Reports
Generation and Identification of Mutations Resulting in Chronic and Age-Related Phenotypes in Mice.
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Blease A. et al, (2018), Curr Protoc Mouse Biol, 8
Novel gene function revealed by mouse mutagenesis screens for models of age-related disease.
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Potter PK. et al, (2016), Nature communications, 7, 12444 - 12444