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Atheroimmunology: keeping the immune system in atherosclerosis in check.
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The extracellular heparan sulfatase SULF2 limits myeloid IFNβ signaling and Th17 responses in inflammatory arthritis.
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Editorial: The interplay between oxidative stress, immune cells and inflammation in cardiovascular diseases.
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Identification of a non-canonical chemokine-receptor pathway suppressing regulatory T cells to drive atherosclerosis
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Position Statement on the Use of Myocardial Strain in Cardiology Routines by the Brazilian Society of Cardiology's Department Of Cardiovascular Imaging - 2023
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Single cell spatial analysis reveals inflammatory foci of immature neutrophil and CD8 T cells in COVID-19 lungs
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Lipid-associated macrophages transition to an inflammatory state in human atherosclerosis, increasing the risk of cerebrovascular complications (Vol 2, Pg 656, 2023)
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Lipid-associated macrophages transition to an inflammatory state in human atherosclerosis, increasing the risk of cerebrovascular complications (vol 2, pg 656, 2023)
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Lipid-associated macrophages transition to an inflammatory state in human atherosclerosis increasing the risk of cerebrovascular complications.
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Modelling systemic risk factors in cardiovascular disease using single-cell biology
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Breaking the macrophage code in atherosclerosis.
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Lipoproteins act as vehicles for lipid antigen delivery and activation of invariant natural killer T cells.
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Neutrophils and emergency granulopoiesis drive immune suppression and an extreme response endotype during sepsis.
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Pyruvate dehydrogenase kinase regulates vascular inflammation in atherosclerosis and increases cardiovascular risk.
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Cell selectivity in succinate receptor SUCNR1/GPR91 signaling in skeletal muscle.
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Three-dimensional transesophageal echocardiographic evaluation of aortic plaque after cerebrovascular event.
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CHECKPOINT ATHERO: developing immune checkpoint-based therapeutics for atherosclerosis.
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A single-cell atlas of immune checkpoint molecules in atherosclerosis in ApoE-/- mice
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Bazioti V. et al, (2023), EUROPEAN HEART JOURNAL, 44
Global deficiency in the inflammatory chemokine receptors 1,2,3 and 5 ameliorates atherosclerosis and selectively modulates aortic myeloid cell phenotype
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