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Calcitonin paracrine signaling controls atrial fibrogenesis and arrhythmia

Journal article

MOREIRA LM. et al, (2020), Nature

Dilated Cardiomyopathy Mutations in Thin Filament Regulatory Proteins Reduce Contractility, Suppress Systolic Ca2+ & Activate NFAT & AKT Signalling

Journal article

Robinson P. et al, (2020), American Journal of Physiology-Heart and Circulatory Physiology

Mavacamten rescues increased myofilament calcium sensitivity and dysregulation of Ca2+ flux caused by thin filament hypertrophic cardiomyopathy mutations.

Journal article

Sparrow AJ. et al, (2020), American journal of physiology. Heart and circulatory physiology

Nitric Oxide and Mechano-Electrical Transduction in Cardiomyocytes.

Journal article

Boycott HE. et al, (2020), Front Physiol, 11

Alpha-tropomyosin mutations in inherited cardiomyopathies.

Journal article

Redwood C. and Robinson P., (2013), J Muscle Res Cell Motil, 34, 285 - 294

The nemaline myopathy-causing E117K mutation in β-tropomyosin reduces thin filament activation.

Journal article

Karpicheva OE. et al, (2013), Arch Biochem Biophys, 536, 25 - 30

The effect of the dilated cardiomyopathy-causing Glu40Lys TPM1 mutation on actin-myosin interactions during the ATPase cycle.

Journal article

Borovikov YS. et al, (2011), Biochem Biophys Res Commun, 411, 496 - 500

The effect of the dilated cardiomyopathy-causing Glu40Lys TPM1 mutation on actin-myosin interactions during the ATPase cycle

Journal article

Borovikov YS. et al, (2011), Biochemical and Biophysical Research Communications, 411, 496 - 500

Functional investigation of beta-tropomyosin mutations that cause congenital skeletal myopathies

Conference paper

Piers A. et al, (2011), NEUROMUSCULAR DISORDERS, 21, S25 - S26

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