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The cellular microenvironment and interplay between cell types are essential for cardiac renewal. Combined single-cell and single-nucleus sequencing, spatial transcriptomics and loss-of-function experiments in constitutively YAP-expressing infarcted hearts reveal a cellular triad and complement signaling that evoke renewal of heart muscle.

Original publication

DOI

10.1038/s44161-024-00426-y

Type

Journal article

Journal

Nature Cardiovascular Research

Publication Date

01/01/2024